Regarding the poisoning of a family that had eaten wild pork and now critically ill, can you post something on secondary poisoning with 1080. Speaking with hunter professionals I gather that this is most likely to be the cause of this Indian family being in intensive care. The toxicology reports are sure to com back as negative for 1080, because that would be too much egg on the governments face.

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From Michelle Read on fb (Michelle's very knowledgeable on 1080 whose opinion I respect) & she comments: 

 "It is unlikely to be botulinum food poisoning. It was too quick after ingestion and there was loss of consciousness. It does not follow the pattern. This is a very serious neurotixin - not at all what one would expect from bacteria. We need to be vigilant for a cover-up of 1080 or other poisonous substance in this instance.

The question needs asking: are pigs are becoming tolerant of the dose? Maybe they ate organ meat, with a high concentration of the toxin - or perhaps it was stored in fat and they ate the fat. 

This is more likely chemical poisonong of some kind - 1080 would be the primary culprit (in a country that did not protect its poison more than people).

So to sum up, if they were affected within an hour from eating and made unconscious then it was not botulinum. It was a neurotoxin and they were poisoned."

Med. J. Aust.; VOL 2 ISS Oct 3 1970, P641-642, (REF 2)

Poisoning due to sodium fluoroacetate (1080)

McTaggart DR

A case is described of poisoning in a boy who ingested wheat previously impregnated with 1080 (sodium fluoroacetate) to poison rabbits. The product causes vomiting, convulsions, coma, respiratory depression and cardiac irregularities. The boy was treated for convulsions with I.V. thiopentone sodium and diazepam. He was resuscitated from cardiac arrest but was left with severe neurological impairment. 1080 is a widely used agricultural poison with cardiotoxic and neurotoxic effects and stingent precautions exist to control its use.

J Toxicol Clin Toxicol 1996;34(6):707-12

Clinical presentation and prognostic factors in sodium monofluoroacetate intoxication.

Chi CH, Chen KW, Chan SH, Wu MH, Huang JJ.

Department of Emergency Medicine, National Cheng Kung University Hospital, Tainan, Taiwan, ROC.

BACKGROUND: The diagnosis of sodium monofluoroacetate intoxication in humans is usually based on a history of ingestion and clinical findings. Although several previous reports have described the clinical course and outcome of patients who ingested this drug, prognostic indicators of short-term survival are not available.

METHODS: A retrospective study of 38 consecutive cases of sodium monofluoroacetate poisoning at the National Cheng Kung. University Hospital, 1988-1993, to analyze the clinical findings and to predict mortality.

RESULTS: Seven of 38 patients (18%) died. The most common symptom was nausea or vomiting (74%). The most frequent ECG finding was nonspecific ST-T and T wave abnormalities (72%). Hypocalcemia (42%) and hypokalemia (65%) were the common electrolyte abnormalities. The clinical and laboratory characteristics were compared for the survival and mortality groups. Significant differences in hypotension, respiratory rate, pulse rate, creatinine, potassium, elevated alanine aminotransferase, pH, PCO2, APACHE II score, and subjective respiratory distress were noted. Discriminant analysis identified hypotension, increased serum creatinine, and decreased pH as the most important predictors of mortality, with sensitivity of 86% and specificity of 96%.

CONCLUSIONS: Hypotension and the early onset of metabolic acidosis and increased serum creatinine are associated with poor short-term survival. These prognostic variables can be useful in the care of patients with suspected sodium monofluoroacetate intoxication. It is suggested that all such patients should be observed intensively for at least 48 h.

PMID: 8941201 [PubMed - indexed for MEDLINE]

Vet Hum Toxicol 2002 Apr;44(2):93-5

Intoxication with sodium monofluoroacetate (compound 1080).

Robinson RF, Griffith JR, Wolowich WR, Nahata MC.

Pediatric Pharmacotherapy/NIH Clinical Research K30 Program, College of Pharmacy, The Ohio State University, Childrens Research Institute, The Central Ohio Poison Control Center, Columbus 43210, USA.

The highly toxic sodium monofluoroacetate (SMFA) was banned as a rodenticide in the U.S. in 1972. We report the first case of intentional ingestion in this country in over 15y. A 47-y-old male was brought to the emergency room status post tonic clonic seizure. At 34 h post ingestion, he responded ony to noxious stimuli and at 48 h, he was unresponsive to painful stimuli, was intubated and placed on a ventilator. Over the following 3 d, he was became minimally responsive to external stimuli with bouts of agitation and hypertension. Two days later he was discharged with no evidence of neurologic sequelae. We report this patient to increase awareness of SMFA toxicity, and its ability to cause anion gap metabolic acidosis.

PMID: 11931513 [PubMed - indexed for MEDLINE]

No one can specifically say what a fatal human dose of 1080 is, however toxicologist  prof. Ian Shaw reported if human sensitivity is the same as dogs then ingesting 0.4mg of 1080  (.4 of 1/1000 of a gram) would be fatal for a 5kg infant.

Apparently information is being deleted by the authorities over the botulism claim in Waikato ... watching still

An update here with health professionals and scientist saying they don't believe it's botulism


by all accounts from the forums is a cover up. All the meat from the family freezer has now been destroyed say no more. Not testing for 1080.

That was very informative thank you Danielle.

Where would we send samples of wild pig to be tested for 1080? Plenty around here who would be into that.


Waikato Regional Councilor Kathy White, having read the finally released medical notes for the Kochumman family poisoned by wild boar in Putaruru, has raised some important questions. Developments with regard to the anomalies surrounding this case have been curious to say the least. Read our previous articles and posts on topic *below.

The questions that need to be asked of both the Waikato DHB and the MPI are:

(1) 1080 was the number one suspect, according to the poisoned patients’ medical notes. The Waikato DHB states that they have tested for 1080. However the date of the urine test was 18 days after hospitalisation, which is outside of the time that 1080 is detectable in urine (it is excreted from urine within seven days of ingestion – See Charles Eason’s Vertebrate Pesticides Toxicology Manual for further details). So why was this test not done within the correct time frame? 

(2) (Incredibly) none of the meat collected by MPI has been tested for 1080. This was confirmed in a meeting with the WDHB yesterday. Why was it was not tested, when the patients’ medical notes say the wild boar meat should be tested for 1080?

(3) Why did they continue to say it was botulism when they knew early on that the symptoms didn’t fully align?

(4) Why has neither the WDHB nor MPI followed up with the person who provided the wild boar meat to Shibu’s family? This information is held by the WDHB. It could be a public health risk as this person has probably supplied meat to others.



Suspected botulism poisoning disproved, family may get ACC after all 4 Jan, 2018 12:24pm

In the letter, which the Herald sighted, Phillips advised that the family had accidental poisoning of an unspecified neurotoxin, the nature of which it was not possible to determine.

She said WDHB had tested for 1080, botulism and other acute neurotoxins like arsenic, mercury, lead, autoimmune antibodies and fish toxin. But they were all negative.

So we are led to believe what MAINSCREAM NEWS TELLS US. 

yes we are Sarah! Is what Kathy is appealing for alternative news sites to call them out. They've clearly been concealing the facts. Even said it wasn't the boar meat after all. They've whisked away all the meat. Doesn't say where that is. Major cover up again. 


Very unfortunate, because although 1080 was suspected early on, they didn’t test for it until EIGHTEEN days later. As the esteemed & so called health professionals would have known at the time, testing for it after 7 days is useless as all presence of 1080 would be gone from the urine & that is according to DoC’s own documentation. So the tests would have been USELESS. (See Charles Eason’s Vertebrate Pesticides Toxicology Manual for further details). This would explain why the family had such difficulty in acquiring their medical notes in the first instance.


Published on Jan 20, 2018
DHB coverup - Politics before patient welfare for wild boar poisoned family

On topic, same interview but some added thoughts & links:

From Waikato Regional Councillor Kathy White:

I've spent the last few weeks reading the medical notes of the Kochumman family that was poisoned through eating wild boar. Here are the things that have resonated with me: (1) the first working diagnosis by the registrar on the day the family was admitted to hospital was "consumption of meat containing poison used for possum/rodent killing ? neurotoxin ? 1080 ? strychnine poisoning which can produce a picture of lactic acidosis, nausea, vomiting, altered mental state and convulsions. Saying this, the differential remains wide, although we do not think this is infection ... I cannot come up with an alternative pathology for raised lactate. May need to see if the police can test the meat for neurotoxins."

If this was the working diagnosis from Day One, why did the authorities not test any of the food for 1080 poison or its metabolites? Why did they not test the patients for 1080 poisoning until 18 days later? The medical notes show doctors were waiting for a 1080 test result. And yet someone made the decision not to do these tests, or to at least delay them. Unfortunately, testing urine for 1080 18 days after hospitalisation does not fit with testing protocol and renders the test unreliable.


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